Volume 1 Issue 1
Jul.  2010
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Article Contents

  • Received Date: 2010-04-27
  • Publish Date: 2010-07-30
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  • [1] Funke H, Assmann G. Strategies for the assessment of genetic coronary artery disease risk[J]. Cur Opin Lipidol, 1999, 10:285-291. doi:  10.1097/00041433-199906000-00011
    [2] Scarabin PY, Arveiler D, Amouyel P, et al. Plasma fibrinogen explains much of the difference in risk of coronary heart disease between France and Northern Ireland. The PRIME study[J]. Atherosclerosis, 2003, 166: 103-109. doi:  10.1016/S0021-9150(02)00309-X
    [3] Behague I, Poirier 0, Nicaud V, et al. β fibrinogen gene polymorphisms are associated with plasma fbrinogen and coronary artery disease in patients with myocardial infarction [J]. Girculation, 1996, 93:440-449 doi:  10.1161/01.CIR.93.3.440
    [4] Carter AM, Mansfield MW, Stickland MH, et al. β fibrinogen gene 455 G/A polymorphism and fibrinogen levels: risk factors for coronary artery disease in subjects with NIDDM [J]. Diabetes Care, 1996, 9:1265-1268. https://care.diabetesjournals.org/content/19/11/1265
    [5] de Maat MPM, Kastelein JJP, Jukema JW, et al. 455G/A polymorphism of the b-fibrinogen gene is associated with the progression of coronary atherosclerosis in symptomatic men: proposed role for an acute phase reaction pattern of fibrinogen[J]. Arterioscler Thromb Vasc Biol, 1998, 18:265-271. doi:  10.1161/01.ATV.18.2.265
    [6] Zito F, Di Castelnuovo A, Amore C, et al. Bel I polymorphism in the fibrinogen b-chain gene is associated with the risk of familial myocardial infarction by increasing plasma fibrinogen levels: a case-control study in a sample of GISSI-2 patients[J]. Arterioscler Thromb Vasc Biol, 1997, 17: 3489-3494. doi:  10.1161/01.ATV.17.12.3489
    [7] Tybjaerg-Hansen A, Agerholm-Larsen B, Humphries SE, et al. A common mutation (G 455-A) in the b-fibrinogen promoter is an independent predictor of plasma fbrinogen, but not of ischemic heart disease: a study of9, 127 individuals based on the Copenhagen City Heart Study[J]. J Clin Invest, 1997, 99:3034-3039. doi:  10.1172/JCI119499
    [8] Wang XL, Wang J, MeCredie RM, et al. Polymorphisms of factor V, factor VⅡ, and fibrinogen genes: relevance to severity of coronary artery disease[J]. Arterioscler Thromb Vasc Biol, 1997, 17:246-251. doi:  10.1161/01.ATV.17.2.246
    [9] Boekholdt SM, Bijsterveld NR, Moons AH, et al. Genetic variation in coagulation and fibrinolytic proteins and their relation with acute myocardial infarction: a systematic review [J]. Circulation, 2001, 104:3063-3068. doi:  10.1161/hc5001.100793
    [10] Lane A, Green F, Scarabin PY, et al. Factor VⅡ Arg/Gln353 polymorphism determines factor VⅡ coagulant activity in patients with myocardial infarction (MI) and control subjects in Belfast and in France but is not a strong indication of MI risk in the ECTIM study[J]. Atherosclerosis, 1996, 119:119-127. doi:  10.1016/0021-9150(95)05638-6
    [11] Doggen CJM, Cats VM, Bertina RM, et al. A genetic propensity to high factor VⅡ is not associated with the risk of myocardial infarctin in men[J]. Thromb Haemost, 1998, 80:281-285. doi:  10.1055/s-0037-1615188
    [12] Domenico G, Carla Russo, Paolo F, et al. Polymorphisms in the Factor VⅡ gene and the risk of myocardial infarction in patients with coronary artery disease[J]. N Engl J Med, 2000, 343:774-780. doi:  10.1056/NEJM200009143431104
    [13] lacoviello L, Di Castelnuovo A, de Knjf P, et al. Polymorphisms in the coagulation factor VⅡ gene and the risk of myocardial infarction[J]. N Engl J Med, 1998, 338:79-85. doi:  10.1056/NEJM199801083380202
    [14] Rosendaal FR, Siscovick DS, Schwartz SM, et al. Factor V Leiden (resistance to activated protein C) increases the risk of myocardial infarction in young women [J]. Blood, 1997, 89:2817-2821. doi:  10.1182/blood.V89.8.2817
    [15] Salomaa V, Matei C, Aleksic N, et al. Soluble thrombomodulin as a predictor of incident coronary heart disease and symptomless carotid artery atherosclerosis in the Atherosclerosis Risk in Communities (ARIC) Study: a case-cohort study[J]. Lancet, 1999, 353:1729-1734. doi:  10.1016/S0140-6736(98)09057-6
    [16] Norland L, Holm J, Zoller B, et al. A common thrombomodulin amino acid dimorphism is associated with myocardial infarction[J]. Thromb Haemost, 1997, 77:248-251. doi:  10.1055/s-0038-1655947
    [17] Thompson SG, Kienast J, Pyke SDN, et al. Hemostatic factors and the risk of myocardial infarction or sudden death in patients with angina pectoris[J]. N Engl J Med, 1995, 332:635-641. doi:  10.1056/NEJM199503093321003
    [18] Ludwig M, Wihn KD, Scheuning WD, et al. Alleleie dimorphism in the human tissue-type plasminogen activator (TPA) gene as a result of an Alu insertion/deletion event [J]. Hum Genet, 1992, 88:388-392. doi:  10.1007/BF00215671
    [19] Ladenvall P, Johansson L, Jansson JH, et al. Tissue-type plasminogen activator-7, 35 1C/T enhancer polymorphism is associated with a first myocardial infarction[J]. Thromb Haemost, 2002, 87: 105-109. doi:  10.1055/s-0037-1612951
    [20] Sobel BE, Woodcock-Mitchell J, Schneider DJ, et al. Increased plasminogen activator inhibitor type 1 in coronary artery atherectomy specimens from type 2 diabetic compared with non-diabetic patients: a potential factor predisposing to thrombosis and its persistence[J]. Circulation, 1998, 97: 2213-2221. doi:  10.1161/01.CIR.97.22.2213
    [21] Margaglione M, Cappucci G, ColaizzoD, et al. The PAI-1 gene locus 4G/5G polymorphism is associated with a family history of coronary artery disease[J]. Arterioscler Thromb Vase Biol, 1998, 18:152-156. doi:  10.1161/01.ATV.18.2.152
    [22] Weis EJ, Bray PF, Tayback M, et al. A polymorphism of a platelet glycoprotein receptor as an inherited risk factor for coronary thrombosis[J]. N Engl J Med, 1996, 334:1090-1094. doi:  10.1056/NEJM199604253341703
    [23] Herrmann SM, Poirier 0, Marques-Vidal P, et al. The Leu 33/Pro polymorphism (PIA1/PIA2) of the glycoprotein Ila (GPⅡa) receptor is not related to myocardial infarction in the ECTIM Study[J]. Thomb Haemost, 1997, 77:1179-1181. doi:  10.1055/s-0038-1656134
    [24] Gonzalez-Conejero R, Lozano ML, Rivera J, et al. Polymorphisms of platelet membrane glycoprotein lba associated with arterial thrombotie disease[J]. Blood, 1998, 92: 2771-2776. doi:  10.1182/blood.V92.8.2771
    [25] The W llcome Trust Case Control Consortium. Genome-wide association study of 14, 000 cases of seven common diseases and 3, 000 shared controls[J]. Nature, 2007, 447: 661-677.
    [26] Hardy J, Singleton A, Genomewide association studies and human disease[J]. N Engl J Med, 2009, 360: 1759-1768.
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