[1]
|
Sekhon MS, Ainslie PN, Griesdale DE. Clinical pathophysiology of hypoxic ischemic brain injury after cardiac arrest:a "two-hit" model[J]. Crit Care, 2017, 21:90. doi: 10.1186/s13054-017-1670-9 |
[2]
|
Harbinson M, Zarshenas S, Cullen NK. Long-term functional and psychosocial outcomes after hypoxic-ischemic brain injury: A case-controlled comparison to traumatic brain injury[J]. PMR, 2017. doi: 10.1016/j.pmrj.2017.04.015.[Epubaheadofprint]. |
[3]
|
Kong X, Guan J, Li J, et al. p66Shc-SIRT1 Regulation of Oxidative Stress Protects Against Cardio-cerebral Vascular Disease[J]. Mol Neurobiol, 2016, 54:5277-5285. http://www.wanfangdata.com.cn/details/detail.do?_type=perio&id=935e2870b9fa7d1cb3b671cc14c119d8 |
[4]
|
Migliaccio E, Giorgio M, Mele S, et al. The p66Shc adaptor protein controls oxidative stress response and life span in mammals[J]. Nature, 1999, 402:309-313. doi: 10.1038/46311 |
[5]
|
Edwards NA, Watson AJ, Betts DH. p66Shc, a key regulator of metabolism and mitochondrial ROS production, is dysregulated by mouse embryo culture[J]. Mol Hum Reprod, 2016, 22:634-647. doi: 10.1093/molehr/gaw043 |
[6]
|
Tomilov AA, Bicocca V, Schoenfeld RA, et al. Decreased superoxide production in macrophages of long-lived p66Shc knock-out mice[J]. J Biol Chem, 2010, 285:1153-1165. doi: 10.1074/jbc.M109.017491 |
[7]
|
Yang CR, Shih KS, Liou JP, et al. Denbinobin upregulates miR-146a expression and attenuates IL-1 beta-induced upregulation of ICAM-1 and VCAM-1 expressions in osteoarthritis fibroblast-like synoviocytes[J]. J Mol Med (Berl), 2014, 92:1147-1158. doi: 10.1007/s00109-014-1192-8 |
[8]
|
El HK, Denoyelle C, Ravaux L, et al. AMPK signaling involvement for the repression of the IL-1 beta-induced group IIA secretory phospholipase A2 expression in VSMCs[J]. PLoS One, 2015, 10:e132498. https://core.ac.uk/download/pdf/52192050.pdf |
[9]
|
Moura MF, Navarro TP, Silva TA, et al. Periodontitis and endothelial dysfunction:periodontal clinical parameters and the levels of salivary markers IL-1beta, TNF-alpha, MMP2/TIMP2 complex, and nitric oxide[J]. J Periodontol, 2017, 88:778-787. doi: 10.1902/jop.2017.170023 |
[10]
|
Jin R, Shen M, Yu L, et al. Adipose-derived stem cells suppress inflammation induced by IL-1beta through down-regulation of P2X7R mediated by miR-373 in chondrocytes of osteoarthritis[J]. Mol Cells, 2017, 40:222-229. https://www.e-sciencecentral.org/articles/pubreader/SC000023673 |
[11]
|
Castejón ML, Rosillo MÁ, Montoya T, et al. Correction:oleuropein down-regulated IL-1beta-induced inflammation and oxidative stress in human synovial fibroblast cell line SW982[J]. Food Funct, 2017, 8:2341. doi: 10.1039/C7FO90020A |
[12]
|
Yamasaki K, Muto J, Taylor K R, et al. NLRP3/cryopyrin is necessary for interleukin-1beta (IL-1 beta) release in res-ponse to hyaluronan, an endogenous trigger of inflammation in response to injury[J]. J Biol Chem, 2009, 284:12762-12771. doi: 10.1074/jbc.M806084200 |
[13]
|
Gao H, Liu L, Zhao Y, et al. Human IL-6, IL-17, IL-1 beta, and TNF-alpha differently regulate the expression of pro-inflammatory related genes, tissue factor, and swine leukocyte antigen class Ⅰ in porcine aortic endothelial cells[J]. Xenotransplantation, 2017, 24:12291. doi: 10.1111/xen.12291 |
[14]
|
Spescha RD, Klohs J, Semerano A, et al. Post-ischaemic silencing of p66Shc reduces ischaemia/reperfusion brain injury and its expression correlates to clinical outcome in stroke[J]. Eur Heart J, 2015, 36:1590-1600. doi: 10.1093/eurheartj/ehv140 |