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微小核糖核酸与糖尿病

杨婧 杨静 李玉秀

杨婧, 杨静, 李玉秀. 微小核糖核酸与糖尿病[J]. 协和医学杂志, 2016, 7(1): 44-47. doi: 10.3969/j.issn.1674-9081.2016.01.009
引用本文: 杨婧, 杨静, 李玉秀. 微小核糖核酸与糖尿病[J]. 协和医学杂志, 2016, 7(1): 44-47. doi: 10.3969/j.issn.1674-9081.2016.01.009

微小核糖核酸与糖尿病

doi: 10.3969/j.issn.1674-9081.2016.01.009
基金项目: 

国家重点基础研究发展计划 973计划

国家重点基础研究发展计划 2014BC542300

北京市科委前沿技术培育项目 Z151100003915077

详细信息
    通讯作者:

    李玉秀 电话:010-69155088, E-mail:liyuxiu@medmail.com.cn

  • 中图分类号: R587.1;Q756

  • [1] Diamond JM. Diabetes running wild[J].Nature, 1992, 357:362-363. doi:  10.1038/357362a0
    [2] DF. One adult in ten will have diabetes by 2030[EB/OL]. http://www.idf.org/media-events/press-releases/2011/diabetes-atlas-5th-edition.
    [3] Grosshans H, Slack FJ. Micro-RNAs: small is plentiful[J]. J Cell Biol, 2002, 156:17-21. doi:  10.1083/jcb.200111033
    [4] Rorsman P, Braun M. Regulation of insulin secretion in human pancreatic islets[J].Annu Rev Physiol, 2013, 75:155-179. doi:  10.1146/annurev-physiol-030212-183754
    [5] Sun LL, Jiang BG, Li WT, et al. MicroRNA-15a positively regulates insulin synthesis by inhibiting uncoupling protein-2 expression[J]. Diabetes Res Clin Pract, 2011, 91:94-100. doi:  10.1016/j.diabres.2010.11.006
    [6] Pullen TJ, da Silva Xavier G, Kelsey G, et al. miR-29a and miR-29b contribute to pancreatic b-cell-specific silencing of monocarboxylate transporter 1(Mct1)[J]. Mol Cell Biol, 2011, 31:3182-3194. doi:  10.1128/MCB.01433-10
    [7] Lee EK, Kim W, Tominaga K, et al. RNA-binding protein HuD controls insulin Translation[J]. Mol Cell, 2012, 45: 826-835. doi:  10.1016/j.molcel.2012.01.016
    [8] Lovis P, Gattesco S, Regazzi R. Regulation of the expression of components of the exocytotic machinery of insulin-secreting cells by microRNAs[J]. Biol Chem, 2008, 389:305-312. doi:  10.1515/BC.2008.026
    [9] Roggli E, Gattesco S, Caille D, et al. Changes in microRNA expression contribute to pancreatic b-cell dysfunction in prediabetic NOD mice[J]. Diabetes, 2012, 61:1742-1751. doi:  10.2337/db11-1086
    [10] Ryu HS, Park SY, Ma DA, et al. The induction of micro-RNA targeting IRS-1 is involved in the development of insulin resistance under conditions of mitochondrial dysfunction in hepatocytes[J]. PLoS One, 2011, 6: e17343. doi:  10.1371/journal.pone.0017343
    [11] Chen HY, Zhong X, Huang XR, et al. MicroRNA-29b inhibits diabetic nephropathy in db/db mice[J]. Mol Ther, 2014, 22:842-853. doi:  10.1038/mt.2013.235
    [12] Frost RJ, Olson EN. Control of glucose homeostasis and insulin sensitivity by the Let-7 family of microRNAs[J]. PNAS, 2011, 108:21075-21080. doi:  10.1073/pnas.1118922109
    [13] Lee H, Jee Y, Hong K, et al. MicroRNA-494, upregulated by tumor necrosis factor-α, desensitizes insulin effect in C2C12 muscle cells[J]. PLoS One, 2013, 8:e83471. doi:  10.1371/journal.pone.0083471
    [14] Yang M, Ye L, Wang B, et al. Decreased miR-146 expression in peripheral blood mononuclear cells is correlated with ongoing islet autoimmunity in type 1 diabetes patients 1miR-146[J]. J Diabetes, 2015, 7:158-165. doi:  10.1111/1753-0407.12163
    [15] Zhou X, Jeker LT, Fife BT, et al.Selective miRNA disruption in Treg cells leads to uncontrolled autoimmunity[J].J Exp Med, 2008, 205:1983-1991. doi:  10.1084/jem.20080707
    [16] Nielsen LB, Wang C, Scrensen K, et al.Circulating levels of microRNA from children with newly diagnosed type l diaetes and healthy controls:evidence that miR-25 associates to residual beta-cell function and glyeaemic control during disease pmgression[J]. Exp Diabetes Res, 2012, 2012: 896362. http://pubmedcentralcanada.ca/pmcc/articles/PMC3518099/
    [17] Berry GJ, Budgeon LR, Cooper TK, et al. The type 1 diabetes resistance locus B10 Idd9.3 mediates impaired B-cell lymphopoiesis and implicates microRNA-34a in diabetes protection[J]. Eur J Immunol, 2014, 44:1716-1727. doi:  10.1002/eji.201344116
    [18] Roggli E, Britan A, Gattesco S, et al.Involvement of microRNAs in the cytotoxic effects exerted by proinflammatory cytokines on pancreatic beta cells[J]. Diabetes, 2010, 59:978-986. doi:  10.2337/db09-0881
    [19] Erener S, Mojibian M, Fox JK, et al. Circulating miR-375 as a biomarker of b-cell death and diabetes in mice[J]. Endocrinology, 2013, 154:603-608. doi:  10.1210/en.2012-1744
    [20] Sebastiani G, Grieco FA, Spagnuolo I, et al. Increased expression of microRNA miR-326 in type 1 diabetic patients with ongoing islet autoimmunity[J]. Diabetes Metab Res Rev, 2011, 27: 862-866. doi:  10.1002/dmrr.1262
    [21] Sebastiani G, Spagnuolo I, Patti A, et al. MicroRNA expression fingerprint in serum of type 1 diabetic patients[J]. Diabetologia, 2012, 55:S48. http://www.researchgate.net/publication/295371075_MicroRNA_expression_fingerprint_in_serum_of_type_1_diabetic_patients
    [22] Zampetaki A, Kiechl S, Drozdov I, et al. Plasma microRNA profiling reveals loss of endothelial miR-126 and other microRNAs in type 2 diabetes[J]. Circ Res, 2010, 107:810-817. doi:  10.1161/CIRCRESAHA.110.226357
    [23] Parrizas M, Brugnara L, Esteban Y, et al. Circulating miR-192 and miR-193b are markers of prediabetes and are modulated by an exercise intervention[J]. J Clin Endocrinol Metab, 2014, 100:E407-E415. http://www.wanfangdata.com.cn/details/detail.do?_type=perio&id=aba10753232cc483dff9366a6d9cd43b
    [24] Ortega FJ, Mercader JM, Moreno-Navarrete JM, et al. Profiling of circulating microRNAs reveals common microRNAs linked to type 2 diabetes that change with insulin sensitization[J]. Diabetes Care, 2014, 37:1375-1383. doi:  10.2337/dc13-1847
    [25] Karolina DS, Tavintharan S, Armugam A, et al. Circulating miRNA profiles in patients with metabolic syndrome[J]. J Clin Endocrinol Metab, 2012, 97:E2271-E2276. doi:  10.1210/jc.2012-1996
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出版历程
  • 收稿日期:  2015-07-22
  • 刊出日期:  2016-01-30

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